Indlela yokutya, utywala kunye nobutyebi obuninzi kunika ingozi kumngcipheko wakho
I-Gout yindlela yesifo se-arthritis esichazwe ngokuhlaselwa ngokukhawuleza, okubandezelekileyo kwintlungu kunye nokuvuvukala kumalungu, ngokuqhelekileyo kuninzi inzwane. Nangona iimeko ezinokuthi zikwazi ukukubeka kwisifo, ezifana ne-genetics okanye izifo zengqondo ezingapheliyo, ezinye ezifana nokutya, utywala kunye nokukhuluphala kunokuba negalelo ngokunjalo.
Ngokubanzi, abantu baya kubakho ukuhlaselwa kokuqala phakathi kweminyaka eyi-30 no-50.
Ngelixa abantu banokuthi banomdla ongaphezu kwabasetyhini, umngcipheko kubasetyhini unokunyuka kakhulu emva kokumisa umva.
Izizathu zokutya
Ngokungafani nezinye iintlobo zesifo samathambo , igulane ibangelwa ukungabikho komzimba kwisifo somzimba kunokuba isistim somzimba. Umngcipheko we-gout uhambelana nemibandela ehlukahlukeneyo-i-genetic, yezokwelapha kunye nendlela yokuphila-ehlangeneyo kunye negalelo lokunyuka kwamazinga e-uric acid egazini, imeko esibhekisela kuyo njenge- hyperuricemia .
Ukutya esikudlayo kudlala indima ebalulekileyo ekuphuhliseni iimpawu ze-gout. Oku kubangelwe inxalenye enkulu kwi-organic composic efumaneka kwiindawo ezininzi ezibizwa ngokuba yi-purine. Xa kudla, i-purine iphulwe ngumzimba kwaye yaguqulwa ibe yimveliso yendalo , i- uric acid . Kwiimeko eziqhelekileyo, kuya kuhlanjululwa ngaphandle kwegazi ngeentso kunye nokuxoshwa komzimba ngomchamo
Ukuba oku kungenzeki kwaye i-acid ye-uric iqala ukuqokelela, ingafaka idizithi ezidibeneyo kunye kunye nokukhokelela ekuhlaselweni kwe-gout.
Ukutya okunye kunye neziphuzo zizinto eziqhelekileyo zezi zinto. Phakathi kwabo:
- Ukutya okuphezulu-purine kubonwa njengengozi enkulu yongxaki. Ezi ziquka ukutya njengezilwanyana zenyama, i-bacon, i-veal, kunye neentlobo ezithile zezilwanyana zaselwandle
- Ubhiya bunzima kakhulu njengoko kuyenziwa ngesilingi se-brewer, isithako esinomxholo ophezulu kakhulu we-purine. Utywala, ngokuqhelekileyo, luyimida njengoko ikhuthaza ukuveliswa kwemilinganiselo ye-uric acid.
- Iziphuzo eziphezulu ze-fructose, eziquka i-sodas kunye neziphuzo ezivuthiweyo ziselo, zingabangela ukuba u-hyperuricemia njengoshukela ogqithiseleyo unciphise ukuphuma kwe-uric acid kwizintso.
Izifo zeGenesis
I-Genetics inokudlala indima ebalulekileyo kwengozi yegout. I-hyperuricemia ye-Hereditary ngumnye umzekelo onjalo, obangelwa yi- SLC2A9 kunye no- SLC22A12 utshintsho olukhokelela ekusebenziseni intsholongwane engasebenzi. Xa kwenzeka oko, iintso zikwazi ukucoca i-uric acid okanye ukubuyisela kwakhona i-uric acid crystals egazini.
Ukungakwazi ukugcina ukulingana phakathi kokukhatyathwa kwe-uric acid kunye nendlela ekhutshwa ngayo kwaye ekugqibeleni iholele ekuxhaseni i-hyperuricemia.
Ezinye izifo zemizimba ezinxulumene nomdlavuza ziquka:
- Ukunganyanzeliseki kwe-fructose
- I-Kelley-Seegmiller syndrome
- Isifo se-Lesh-Nyhan
- Izifo zesifo sezintso zesifo se-Medullary
Izizathu Zonyango
Kukho imeko ezithile zezokwelapha ezingakunqanda ukutshiza. Abanye bachaphazela ngqo okanye ngokungathanga ngqo umsebenzi wokuguqulwa komsebenzi, ngelixa ezinye zibonakaliswa ngempendulo engavumelekanga eyenziwa ngabanye ososayensi banokukhuthaza ukuveliswa kwe-uric acid.
Ezinye zezinto eziqhelekileyo zempilo yengozi zibandakanya:
- Izifo zesifo esingapheliyo
- Ukukhubazeka kwentliziyo
- Sikashukela
- Hemolytic anemia
- Uxinzelelo oluphezulu (uxinzelelo lwegazi oluphezulu)
- I-Hypothyroidism (umsebenzi ophantsi we-thyroid)
- Lymphoma
- Psoriasis
- I-Psoriatic arthritis
Ezinye iziganeko zonyango ziyaziwa ngokubangela ukuhlaselwa kwe-gout, kubandakanywa ukulimala ngokubambisana, ukusuleleka, ukuhlinzwa kwangoku kunye nokutya kokuphazamiseka (okokugqibela okungakwandisa ukukhula kwe-uric acid ngenxa yokunciphisa ngokukhawuleza komthamo womzimba).
Izizathu ZeMithi
Amanye amachiza anxulumene ne-hyperuricemia nokuba ngaba i-diuretic effect (ukwandisa umxube we-uric acid) okanye uchithe ukusebenza komnxeba. Amacala athile aqhelekileyo afaka:
- I-Aminophylline (sebenzisa ukunyanga i-bronchitis engapheliyo)
- I-Cyclosporine (isichengululo sokukhuselana nomzimba)
- I-Ethambutol (isetyenziselwa ukunyanga isifo sofuba)
- Lasix (furosemide)
- I-Levodopa (esetyenziselwa ukuphatha isifo sikaParkinson)
- I-aspirin ye-dose ephantsi (isetyenziselwa ukunciphisa umngcipheko wokuhlasela kwentliziyo)
- I-Niacin (i-vitamin B3)
- I-thiazide diuretics (esetyenziselwa ukunyanga uxinzelelo lwegazi okanye isifo senhliziyo)
Izinto zokuPhila kweengozi
Iinqunto ozenzayo ebomini zidlala indima enkulu kumngcipheko we-gout njengemibandela ongenakuyilawula, njengeminyaka okanye ubulili. Akunakuphelisa ngokupheleleyo umngcipheko wakho, kodwa unokuchaphazela ukuba kaninzi kangakanani ukuhlaselwa.
Ukunyanya
Oyintloko phakathi kwezi zinto zixhalabisa. Ngokwakhe, ukunyameka komzimba kunciphisa ukususwa kwe-uric acid emzimbeni. Kwaye, nangaphezulu xa ulinganisa, lukhulu lo monakalo uya kuba.
Ukukhushulwa kwe-insulin ngenye yezinto eziqhubela phambili kule nto. Ukuba ukhuluphele okanye ukhuluphele, umzimba wakho uvelisa ngaphezulu kwe-insulin. Amanqanaba aphakamileyo e-insulin ekhokelela ekudakaleni kwemfucuza ekukhohliseni umonakalo ekhokelela kumazinga aphezulu e-uric acid.
Ucwaningo luka-2015 lufumene ulungelelwano oluchanekileyo phakathi komlenze womntu kunye nomngcipheko we-gout. Ngokwabaphandi, phakathi kwabantu abanomdlavuza, abo banamafutha aphezulu esiswini banomngcipheko we-47.4 we-intlekiso xa kuthelekiswa nalabo abanezintlu eziqhelekileyo abanomngcipheko we-27.3 wepesenti. Oku kungakhathaliseki ukuba ngumzimba we-mass body index (BMI), ebonisa ukuba amafutha amaninzi esiwaphetheyo, umngcipheko omkhulu weempawu.
Ezinye Izinto
Ukususela kwimibono yokulawulwa kwempilo, ezininzi izinto ezinxulumene nezifo ezingapheliyo ezifana nesifo seswekile nesifo se-cardiovascular zixhunyiwe kwi-gout. Ezi ziquka:
- I-fatcer visceral excess (amafutha esiswini)
- Uxinzelelo lwegazi oluphezulu (ngaphezu kwe 130/85 mmHg)
- I-LDL ephezulu ('okubi') ye-cholesterol kunye ne-HDL ephantsi ("okuhle") i-cholesterol
- I-triglycerides ephakamileyo
- Ukuxhathisa kwe-insulin
- Ukusetyenziswa rhoqo kotywala
- Indlela yokuphila
> Imithombo:
> Hanier, B; UMatheson, E. noWilke, T. "Ukuxilongwa, unyango kunye nokukhuselwa kweGout." Am Fam Physician. 2014; 90 (12): 831-836.
> Richette, P. noBarden, T. "Gout." Lancet. 2010; 375 (9711): 318-28. INGXELO: 10.1016 / S0140-6736 (09) 60883-7.
> Rothenbacher, D .; Kleiner, A. Koenig, W. et al. "Ubudlelwane phakathi kwamaCytokines anokukhuphaza kunye namazinga e-Uric Acid neeMiphumo eziPhezulu zeMiphumo yeZifo kwizigulane ezineSifo seCononary Heart Disease." PLoS One. 2012; 7 (9): e45907. INGXELO: 10.1371 / iphephandaba.pone.0045907.
> Roughley, M .; Belcher, J; Mallen, C. et al. "I-Gout kunye nomngcipheko wesifo esingasigxina nesifo se-nephrolithiasis: uhlalutyo lweemeta-uphando lwezifundo." I- Arthritis Res Ther. 2015; 17 (1): 90. INGXELO: 10.1186 / s13075-015-0610-9.