Uric acid iveliswa xa umzimba uphula i-purines (i-purines inika inxalenye yendlela yamakhemikhali ethu). Iingqayi zombini zekhabhoni-nitrogen isiseko, i-adenine, kunye ne-guanine, zihlambulukile. Iipilisi zikhonza njengendlela yamandla kwiiseli kwaye ziyimfuneko ekuveliseni i-DNA kunye ne-RNA, iiproteni, isitashi, imimiselo yee-enzyme kunye nesalathisi.
Imveliso ye-uric acid ihambelana nokuhlanganiswa kwe-purines ngaphakathi komzimba kunye nokuxhamla i-purine intake.
Ngamanye amagama, i-purines ifumaneka kuzo zonke izicubu zomntu kwaye zifumaneka kwiindawo ezininzi zokutya . Xa amaseli efa emzimbeni wethu, i-purines ikhutshwa. I-acid ye-Uric ngumveliso wokuphela okanye isifo se-purabolism yabantu. Ukupheliswa kwe-uric acid ukusuka emzimbeni kuxhomekeke kwi-excretion. Yiyo le mali - phakathi kwemveliso ye-uric acid kunye ne-excretion-leyo inquma izinga le-urum urate (izinga le-uric acid egazini).
Amanqanaba eSirate Urate
Uninzi lwe-uric acid uhlahlela egazini kwaye uhambela kwiintso ezizocatshungulwa kumchamo. Ngokuqhelekileyo, abantu bagcina isilinganisi se-urum urate level phakathi kwe-4 no-6.8 mg / dl, kunye ne-body body uric acid ye-1,000 mg. Abantu abavelisa uric acid kakhulu okanye abangakwazi ukuphelisa uric acid okwaneleyo baye baphakamisa amanqanaba e-serum urate. Inqanaba eliphezulu le-uric acid egazini libizwa ngokuba yi- hyperuricemia . Ukukhutshwa ngokweqile okanye ngaphantsi kwe-uric acid, okanye ukudibanisa kokubili, kunokukhokelela ekuxhaseni i-hyperuricemia.
Abantu abanezinga eliphezulu le-uric acid egazini banokuvelisa isifo sesifo okanye igulane ngokuphuhliswa kwamakristal kumalungu. Akunjalo bonke abantu abane-hyperuricemia bavelisa i-gout.
Kubalulekile ukugcina inqanaba le-serum urate evamile. Ukuhlolwa kwegazi yindlela enye yokujonga izinga lakho le-uric acid. Inokukhangela kwakhona kwisampuli yomchamo.
Ngokufanelekileyo, i-serum uric acid kufuneka ibe ngu-6.0 mg / dl okanye iphantsi. Umntu onesilinganisi se-uric acid esingu-6.8 mg / dl okanye ngaphezulu uhlelwa njengobuncwane.
I-Hyperuricemia ingenzeka ngeemeko ezahlukeneyo, kubandakanywa:
- acidosis
- utywala
- nemiphumo emibi yekhemotherapy
- gout
- isifo samarthritis esingapheliyo
- iswekile
- hypoparathyroidism
- khokela utyhefu
- leukemia
- isifo sesifo
- ukuphelelwa kweengtso
- polycythemia vera
- ukutya okunotyebileyo
- i-toxemia ehlobene nokukhulelwa
Amanqanaba aphantsi e-uric acid angabandakanywa:
- ukutya okuphantsi kwe-purine
- Fanconi syndrome
- ukugcinwa kwe-hormone ye-antidiuretic
- Isifo seWilson
Ngaphantsi
Kubalulekile ukwazi isilinganisi sakho se-uric acid, akukho hlukile kunokuba kubalulekile ukwazi i-cholesterol yakho okanye i-blood glucose level. Abantu abanembali ye-gout kufuneka babe nezinga le-uric acid eyenziwa rhoqo kwiinyanga ezi-6 ukujonga ukuba ihlala phantsi kwe-6.0 mg / dl. Izigulane zeGout zingadinga unyango ngamachiza kunye nokuguquka kwendlela yokuphila ukuze kugcinwe i-uric acid kwizinga elifanelekileyo.
Imithombo:
Uric Acid - Igazi. MedlinePlus.
https://www.nlm.nih.gov/medlineplus/ency/article/003476.htm
Malunga neAric Acid. Gout & Uric Acid Society Society.
http://gouteducation.org/patient/what-is-gout/uric-acid/
I-Kelley's Bookbook yeRheumatology. I-ninth edition. Elsevier. Isahluko 94: I-Etiology ne-Pathogenesis ye-Hyperuricemia kunye neGout.
I-Purine nePyrimrimidine Metabolism. Hlukana.
https://www.diffen.com/difference/Purines_vs_Pyrimidines