Kukho izimbalwa ezinokubangela ukuhluleka kwentliziyo, zonke ezo zinomxholo ofanayo wokunciphisa intliziyo. Ukungaphumeleli kwintliziyo kunokubangelwa yizinto ezinjenge-cardiovascular issues ezifana nokuhlasela kwentliziyo, isifo se-coronary disease (umonakalo ngaphakathi kwemida yegazi yentliziyo), kunye nomfutho wegazi ophezulu (uxinzelelo lwegazi), kunye nezinye izifo kunye nezimo, ezifana nesifo sikashukela kunye ukunyanya.
Izinto zokuphila, ezifana nokutshaya nokungahambi komsebenzi, zidlala indima ebalulekileyo, njengoko zidla ngokubangela ezinye zezixhala. Imfuyo yezofuzo, i- hypertrophic cardiomyopathy , yinto eqhelekileyo ngokufanayo.
Uxinzelelo kwiintliziyo zentliziyo ngexesha elide lichithezela ukuhamba kwegazi ngokugqithiseleyo ukuya kwinqanaba lokuba kukho i-buildup of fluid entliziyweni nasemiphungeni, kwaye ekugqibeleni, ukugqithisa okwedlulele kwimida. Iimpawu ezifana nokuphefumula, ukukhathala, kunye ne-edema (ukuvuvukala kwezandla kunye neenyawo) yiphumo lomsebenzi wenhliziyo obuthathaka obonakalisa ukuhluleka kwentliziyo.
I-Cardiovascular
Kwizinto ezibangela ukungaphumeleli kwentliziyo, ezibalulekileyo ziimeko eziphambili zentliziyo. Abanye bavame ukuhlangana kunye kwaye kunokubangela omnye komnye. Ngokomzekelo, uxinzelelo lwengcinezelo luba negalelo kwi-coronary artery disease, ekhokelela ekuhlaselweni kwentliziyo.
Imiba exhaphakileyo yentsholongwane yempilo eyenza ukuhluleka kwentliziyo kuku:
Uxinzelelo lwengqondo: Uxinzelelo lwexinzelelo luyimbangela ebangela ukuba amadoda namabhinqa. Ukuma kwexesha elide uxinzelelo lweengxowankulu kuncedisa kwisifo se-coronary disease, esona sizathu esibangeleyo sokuhlasela kwentliziyo (umonakalo ohambelanayo uyanciphisa intliziyo, ngamanye amaxesha kubangela ukuhluleka). Uxinzelelo lwengqondo luphela luba negalelo ekuphelelweni kwentliziyo ngenxa yokuba, xa intliziyo igxobhozela uxinzelelo ophezulu kwiminyaka, izihlunu zingaphantsi kakuhle.
I-CAD (i-coronary disease artery): Imithambo ye-coronary yimizila yegazi ehambisa intliziyo ngegazi kunye ne-oksijini enegazi. I-CAD ichaza inkqubo apho ngaphakathi kwimiba ye-coronary ibe yincinci, iqinile, kwaye ingavumelekanga. Le mijelo yegazi engafanelekiyo ixhomekeke ekuqongeni kwe-cholesterol, i-debris kunye negazi. Ekugqibeleni, zinokuthi ziphazamiseke ngamacandelo egazi, ezibangela ukuhlasela kwentliziyo.
MI (i-myocardial infarction): I-infyoction ye-myocardial yi-heart attack. Oku kwenzeka xa i-clot yegazi ikhusela ngokupheleleyo enye okanye eminye imitha yeeronari, iphazamise ukuhamba kwegazi kwinqanaba lentliziyo. Xa iindawo zeentliziyo zithintela igazi, zingaze zisebenze ngendlela efanayo, ziba buthathaka kumaqela achaphazelekayo yintliziyo. Oku kwenza ukuba intliziyo yokupompoza isebenze kangangoko, ikhokelela ekuphelelweni kwentliziyo.
Iimfumba zentliziyo ezibuthakathaka zithatha ukunweba kwaye, ngenxa yoko, amagumbi okupompoza entliziyweni, ngokuqhelekileyo i- ventricle ekhohlo , iyancipha (yandiswe). I-ventricle ehlanjulweyo inomthamo omkhulu wegazi, ngoko ke igazi elingakumbi linokukhishwa ngenhliziyo ephosakeleyo.
Ukongezelela, iingcinezelo ngaphakathi kwintliziyo zanda, kubangela ukuba umbane uphindele kwimiphunga, kuvelise ukudibanisa kwemiphunga.
Iimpawu zentliziyo ye- cardiac (iintsholongwane ezingapheliyo), ezinokubangela ukongela ubomi, ziqhelekile kubantu abane-cardiomyopathy.
I-aortic valve stenosis : I- aortic stenosis iyanciphisa i-aortic valve, eyandisa kakhulu uxinzelelo kunye noxinzelelo kwi-ventricle yentliziyo. Oku kubangela ukuhluleka kwentliziyo ngexesha.
Ukuhluleka kwentliziyo ye-Diastolic: Ukuhluleka kwentliziyo ye- diastolic , umsebenzi wentliziyo uyawohloka ngenxa yokuba imisipha yentliziyo iba nzima kodwa ingabi mkhulu, njengakwezinye iintlobo zokwehluleka kwentliziyo. Olu lukhuni luvimbela intliziyo ukuba ikhululeke ngendlela efanele, yenza kube nzima ukuba igcwalise ngokwaneleyo ngegazi phakathi kweentliziyo.
Ngenxa yoko, inani legazi elipompozwe ngentliziyo nganye liyancitshiswa, okubangelwa ukukhathala nokunyamezela ukunyamezela. Igazi elingenako ukuzalisa intliziyo "libuyiselwa" kwimiphunga, kuvelisa ukudibanisa kwempompo . Ukukhubazeka kwe-Diastolic kuthatha ukuvela kubantu abadala, ngakumbi abafazi.
Izimo zentliziyo yengane: Izifo zentliziyo yobuntwabongqongqele, njengentliziyo ye-anatomiki okanye iimpazamo zepulmoni, ukungasebenzi kwemigodi, kunye nokungaqhelekanga okuchaphazela isakhiwo se-aortic, kubangela ukuhluleka kwentliziyo. Abantwana abaselula banokuphuhliswa kwentliziyo kwiminyaka embalwa ukuba le miqathango ayiphathwa. Ukulungiswa kokuvuthwa okanye ukutsalwa kwintliziyo kudlalwa njengento yokwelapha ukunciphisa ubunzima obugqithiseleyo kwiintliziyo zentliziyo, ngaphezu kwempembelelo yesiphene esisisiseko.
Systemic
Nangona kucacile ukubona indlela izifo kunye neemeko ezihambelana ngqo nentliziyo zikhokelela ekuphelelweni kwentliziyo, kukho ezinye izizathu ezingabonakali.
Isifo sikashukela: Isifo sikashukela ngokwawo asivelisi ngokuthe ngqo ukusilela intliziyo, kodwa sinceda kwiimeko ezenzayo, ezifana ne-CAD kunye ne-MI. Abantu abanesifo sikashukela nabo baneziganeko eziphezulu zexinzelelo.
I-Chemotherapy: Amachiza athile anamandla asetyenziswa kunyango lomhlaza, ngakumbi i- Adriamycin (doxorubicin) , kunokubangela ukuba inetyhefu yesifo ekhokelela ekuphelelweni kwentliziyo. Ngokungafani neminye yemingcipheko echazwe apha, ethatha ixesha elide ukuba nefuthe, i-chemotherapy ingenza ngokukhawuleza.
Ukubeleka: I- postpartum cardiomyopathy yintlobo yesifo senhliziyo esichaphazelekayo. Nangona le meko ihlala isombulula ngonyango olukhuselekileyo, ivelisa umngcipheko ophezulu wexesha elide wokuphuhliswa kwentliziyo ngexesha elizayo, ngakumbi ngokukhulelwa kwangaphambili.
Ukuxinezeleka okukhulu: Ukuxinezeleka kwengqondo , okubizwa nangokuthi "i-heart distil syndrome," yindlela yokuphazamiseka kwentliziyo ngokukhawuleza, eyabangela ukukhathazeka okukhulu. Nangona le meko ibonakala kwindoda yesini, ixhaphake kakhulu kwabesetyhini kwaye inokuthi ihambelane ne- angina encinci , imeko eyenzeka kakhulu kwabesetyhini.
I-apnea yokulala : imeko ebonakalayo ngokuphazamiseka emfutshane kokuphefumula ngexesha lokulala. Nangona i-apnea yokulala ayidla ngokufa, i-apnea yokulala yangexesha elide elingapheliyo inegalelo kwiimeko ezininzi zempilo, ezinjengokukhubazeka kwentliziyo. Indlela echanekileyo yesi sixhobo asicaci ngokupheleleyo.
Genetic
Ukuqondwa kweengcambu zezofuzo zesifo senhliziyo kukhula. Impembelelo yeHereditary kwiindlela zakho zokuphuhliswa kwentliziyo yinkxalabo, kodwa kunjalo yimeko ezuze ilifa eyaziwa ngokuba yimizimba, ebizwa ngokuba yi-hypertrophic cardiomyopathy. Kwaye, njengoko kukhankanywe, iimeko zeentliziyo zengane zendalo zingakhokelela ekuphelelweni kwentliziyo.
I-genetic predisposition : Iingcambu ezingama-100 ziye zachongwa njengento ehambelana nokuhluleka kwentliziyo, kwaye kuya kubonakala ngokucacileyo ukuba ukungaphumeleli kwintliziyo, ngokuqhelekileyo, kukufa. Nangona kunjalo, ikhonkco phakathi kweentlobo zeempawu, isifo sokuhlaziya isifo, kunye neengqikembe ezithile zendalo azizange zenziwe kakuhle.
I-cardiomyopathy ye-Hypertrophic : Le meko yezofuzo ibonakaliswa ngokunyanyiswa kwesisu senhliziyo. Inokuqala ukuvelisa iimpawu ebuntwaneni, ebuntwaneni, okanye emdala. Ubunzima bubaphazamisa ukuzaliswa kwentliziyo kwaye kunokukhokelela kwiziqwenga zokuphefumula okungapheliyo, ngakumbi ngexesha lokuzivocavoca. Ukunyanyiswa kwesifo senhliziyo kunokubangela ukuphazamiseka kwi-ventricle ekhohlo, efana neyabonwa nge- aortic stenosis . Abanye abantu abane-hypertrophic cardiomyopathy banomngcipheko ophezulu wokufa ngokukhawuleza.
Iimpawu ziquka ukuphefumula okufutshane, ukungabi nantliziyo yezinga elingaqhelekanga, ukuphazamiseka ngokukhawuleza, kunye nokufa ngokukhawuleza. Utyando oluthengwayo olunjengama-pacemakers kunye neenkqubo zokuvula iindlu zeentsimbi zentliziyo zinganciphisa ezinye zengcinezelo kunye nokudibanisa entliziyweni, ukuphucula amathuba okuhlala ixesha elide.
Indlela yokuphila
Ngokuqhelekileyo, izinto zokuphila zenza igalelo kwezinye zeemeko zentliziyo eziphambili kwaye zibangele ukungaphumeleli kwentliziyo, kungekhona ngokuthe ngqo kwintliziyo yokuhluleka.
Ukunyaniseka: Abantu abadala kunye nabaselula abasweleyo banomngcipheko ophezulu wokuphulukana kwentliziyo. Oku kuncinci ngenxa yokuba intliziyo kufuneka isebenze nzima ukuba unikeze umzimba ngegazi elaneleyo xa unzima kakhulu. Ukunyameka kwimeko yengozi yesifo sikashukela, i-hypertension, ne-CAD, yonke into ekhokelela ekuphelelweni kwentliziyo.
Ukubhema nokusetyenziswa kweziyobisi : Ngokuqhelekileyo, kuyaziwa ukuba ukutshaya ngenye yezinto ezinobungozi kwi-MI, kwaye oku kuncinci ngenxa yokuba umkhwa unika i-CAD. Iziyobisi, ezifana ne-methamphetamine, ziye zadibaniswa nokuhluleka kwentliziyo.
Indlela yokuphila yokuhlala ngokwexesha elidlulileyo: Ukungahlali ixesha elide, okuqhelekileyo kuchazwa ngokuba uhleli ixesha elide ixesha eliqhelekileyo, liboniswe ukwandisa amathuba okwehluleka kwentliziyo. Ukunyakaza okungakumbi kwansuku zonke kunye nokuzilolonga rhoqo, oku kuchazwe njengeeseshoni ezine ukuya ezintlanu ngeveki, kuye kwadibaniswa nesigxina esingaphantsi kwenhliziyo.
I-cardiac kunye nempilo yokuphefumula : Oku kuchaza ubuchule benhliziyo nemiphunga ukuba isebenze ngokufanelekileyo. Unokuphuhlisa ukuqina komzimba wakho ngokuthatha rhoqo inxaxheba kwimisebenzi eyenza intliziyo yakho ibe yintengo, eqinisa intliziyo yakho ngokuhamba kwesikhathi, uvumele ukuba batshayele ngamandla amakhulu. Unokuphucula impilo yakho yokuphefumula ngokuthatha inxaxheba kwimisebenzi eyenzela ukuba uphefumle ngokukhawuleza, eqeqesha imiphunga yakho ukuze ithathe i-oksijini emzimbeni wakho ngokufanelekileyo.
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